Гравиметрическим, электрохимическим и физико-химическим (рентгенофазовый анализ, УФ- и ИК-спектроскопия) методами изучен процесс коррозии стали Ст. 3 в воде с добавкой хлористого (дигидразид семикарбазиддиуксусной кислоты) никеля (II) тригидрата. Приводятся сравнительные результаты влияния на скорость коррозии комплексного соединения, а также лигандов - дигидразида семикарбазиддиуксусной кислоты и гидразина-1,1-диуксусной кислоты. Установлено существенное подавление коррозии стали за счет адсорбции ингибитора на корродирующей поверхности и комплексообразования ионизированного железа с продуктами распада ингибитора с последующим включением нерастворимых комплексов в покровный защитный слой.
Gravimetric, electrochemical and physicochemical (the X-ray phase analysis, UV-and IR-spectroscopy) methods were applied to study the process of corrosion of St. 3 steel of in water with addition of chloride (dihydrazide of semicarbazidiacetic acid) nickel (II) of a trihydrate. Comparative investigation of the influence of a complex compound and the mentioned ligands on the corrosion rate has been performed. It was found that there is an essential suppression of the steel corrosion due to the adsorption of the inhibitor on a corrosive surface and to the complex formations of the ionized iron with products of disintegration of the inhibitor, with the subsequent inclusion of insoluble complexes in an integumentary protective layer. Gravimetric, electrochemical and physicochemical (the X-ray phase analysis, UV-and IR-spectroscopy) methods were applied to study the process of corrosion of St. 3 steel of in water with addition of chloride (dihydrazide of semicarbazidiacetic acid) nickel (II) of a trihydrate. Comparative investigation of the influence of a complex compound and the mentioned ligands on the corrosion rate has been performed. It was found that there is an essential suppression of the steel corrosion due to the adsorption of the inhibitor on a corrosive surface and to the complex formations of the ionized iron with products of disintegration of the inhibitor, with the subsequent inclusion of insoluble complexes in an integumentary protective layer.